INNOGLY WG2 minisymposium: Autophagy

INNOGLY WG2 minisymposium

June 21, 3 pm – 5 pm Central European Time

 

Keynote at 3 pm CET

David Sleat, Associate Professor, Center of Advanced Biotechnology and Medicine, Rutgers University, New Jersey, USA

Lysosomal proteomics and human disease

Examples of publications by David Sleat:

Analysis of Brain and Cerebrospinal Fluid from Mouse Models of the Three Major Forms of Neuronal Ceroid Lipofuscinosis Reveals Changes in the Lysosomal Proteome.

Sleat DE, Wiseman JA, El-Banna M, Zheng H, Zhao C, Soherwardy A, Moore DF, Lobel P. Mol Cell Proteomics. 2019 Nov;18(11):2244-2261. doi: 10.1074/mcp.RA119.001587.

 

Inducible transgenic expression of tripeptidyl peptidase 1 in a mouse model of late-infantile neuronal ceroid lipofuscinosis.

Nemtsova Y, Wiseman JA, El-Banna M, Lobel P, Sleat DE. PLoS One. 2018 Feb 6;13(2):e0192286. doi: 10.1371/journal.pone.0192286. eCollection 2018.

 

 

Talks by INNOGLY members, 4 – 5 pm CET:

Massimo Aureli, University of Milan, Italy:

Involvement of lysosomal impairment in the onset of neuronal damage
 

Ole Kristian Greiner Tollersrud, Arctic University of Norway, Tromso:

Trimming of N-glycans in the lysosomes: A novel processing pathway
 

Mattan Hurevich and Shlomo Yitzchaik, The Hebrew University of Jerusalem, Israel:

Studying glycan interactions using electrochemical impedance spectroscopy

 

 

Register to the minisymposium by June 10 to:

eeva-liisa.eskelinen@utu.fi.